Myocardial Infarction (MI): Evidence-Based Nursing Guide + NORCET-Style MCQs
Aligned with Indian nursing practice & exam patterns. Includes interactive MCQs with rationales, Table of Contents, sharing buttons, and FAQ schema.
Introduction
Myocardial infarction (MI) is irreversible necrosis of cardiac muscle due to prolonged ischemia. For Indian nurses, rapid recognition, protocol-driven action, and accurate monitoring directly impact survival and recovery. This guide summarizes practical, evidence-based points for assessment, immediate care, monitoring, complications, discharge education and secondary prevention—followed by NORCET-style MCQs.
Pathophysiology & Risk Factors
- Mechanism: Atherosclerotic plaque rupture → platelet activation → thrombus → coronary occlusion.
- Types: STEMI (transmural), NSTEMI (subendocardial), silent MI.
- Risk factors (modifiable): Hypertension, diabetes, dyslipidemia, smoking, obesity, sedentary lifestyle, high salt/sugar/trans-fat intake.
- Risk factors (non-modifiable): Age, sex, family history, early CAD in relatives.
Clinical Presentation & Diagnosis
- Symptoms: Central chest pain/pressure (radiation to left arm/jaw/back), dyspnea, diaphoresis, nausea/vomiting, fatigue; atypical in women, elderly, diabetics.
- ECG: ST-elevation in contiguous leads (STEMI); ST-depression/T-inversion (ischemia/NSTEMI); new LBBB may indicate MI.
- Cardiac biomarkers: Troponin I/T rise 3–6 h, peak 12–24 h, remain elevated up to 7–14 days; CK-MB useful to detect reinfarction.
- Other: Echo (wall-motion), CXR, lipid profile, RBS/HbA1c, renal function, electrolytes.
Nursing Management (Indian Context)
- Immediate actions: Alert team, attach monitor, IV access, aspirin loading per protocol, SL nitroglycerin (if no contraindication), analgesia. Prepare for reperfusion—PCI (door-to-balloon ≤ 90 min). If PCI delay expected and eligible, fibrinolysis (door-to-needle ≤ 30 min).
- Oxygen: Give if SpO₂ < 90% or respiratory distress/hypoxemia; avoid routine oxygen when not indicated.
- Monitoring: Vitals, rhythm/ECG, troponin trend, urine output, pain scale, mental status; watch for arrhythmias, acute HF, shock, mechanical complications.
- Medications (as prescribed): DAPT, anticoagulant, statin, beta-blocker (avoid in shock/bradycardia/asthma), ACEI/ARB, nitrates, analgesia.
- Support & education: Anxiety reduction, clear communication, early cardiac rehab (Phase I once stable).
- Discharge & secondary prevention: Adherence to DAPT (as advised), high-intensity statin, BP/DM control, tobacco cessation, diet, graded activity, rehab follow-up.
NORCET-Style MCQs on MI (Click “Show Answer”)
- New left bundle branch block only
- ST-segment depression in V1–V3
- ST-segment elevation in contiguous leads
- Peaked T waves only
Correct: C. STEMI is diagnosed by ST-elevation in anatomically contiguous leads.
- Give SL nitroglycerin and observe for 1 hour
- Prepare for emergent PCI and expedite transfer to cath lab
- Send lipid profile first
- Begin ambulation
Correct: B. Early reperfusion is time-critical; activate the PCI pathway.
- CK-MB
- Myoglobin
- Cardiac troponin I/T
- LDH
Correct: C. Troponins rise 3–6 h, peak 12–24 h, remain elevated up to 1–2 weeks.
- ≤ 120 minutes
- ≤ 90 minutes
- ≤ 60 minutes
- ≤ 45 minutes
Correct: B. Benchmark target ≤ 90 minutes.
- ≤ 60 minutes
- ≤ 45 minutes
- ≤ 30 minutes
- ≤ 15 minutes
Correct: C. Aim within 30 minutes of hospital arrival.
- SpO₂ ≥ 98%
- All suspected MI, routinely
- SpO₂ < 90%, or respiratory distress/hypoxemia
- Only after PCI
Correct: C. Avoid routine oxygen in non-hypoxemic patients.
- V1–V4
- V5–V6, I, aVL
- II, III, aVF
- aVR only
Correct: C. These leads reflect inferior wall involvement.
- Anterior STEMI with hypertension
- Right ventricular infarction with hypotension or recent sildenafil use
- Mild headache
- Normal BP
Correct: B. Risk of profound hypotension.
- Ventricular septal defect
- Acute severe mitral regurgitation with pulmonary edema
- Pericardial tamponade
- Bradyarrhythmia
Correct: B. Mechanical complication 2–7 days post-MI.
- 0–1 hour
- 3–6 hours
- 24–48 hours
- 72 hours
Correct: B. Rise 3–6 h, peak 12–24 h, persist up to 1–2 weeks.
- Clopidogrel alone
- Aspirin loading dose (chewed) as per protocol
- Warfarin
- Wait until troponin positive
Correct: B. P2Y12 inhibitor is then added (DAPT) per protocol.
- HR 88/min, BP 138/82 mmHg
- Mild anxiety
- Cardiogenic shock, severe bradycardia, or SBP < 90 mmHg
- Anterior STEMI
Correct: C. Avoid in shock, marked bradycardia/hypotension, acute decomp HF, severe asthma.
- Aspirin
- High-intensity statin
- Loop diuretic
- Nitrate
Correct: B. Statins reduce LDL-C and stabilize plaque.
- Immediately after thrombolysis while unstable
- In-hospital once clinically stable (often within 24–48 h)
- 6 months after MI
- Only after stent removal
Correct: B. Early supervised mobilization & education improve outcomes.
- Coronary spasm without atherosclerosis
- Venous embolism
- Atherosclerotic plaque rupture with thrombus
- Myocarditis
Correct: C. Plaque rupture with thrombosis accounts for most acute MIs.
Frequently Asked Questions (FAQ)
MI is death of heart muscle due to prolonged ischemia from reduced blood flow, usually caused by a blocked coronary artery.
STEMI shows ST-segment elevation on ECG and indicates full-thickness ischemia; NSTEMI lacks ST-elevation but has positive troponin with ischemic symptoms/ECG changes.
Oxygen is recommended if SpO₂ is below 90% or if the patient has respiratory distress/hypoxemia. Routine oxygen in non-hypoxemic patients is not recommended.
PCI door-to-balloon ≤ 90 minutes; fibrinolysis door-to-needle ≤ 30 minutes when PCI is delayed and the patient is eligible.
Yes—fatigue, breathlessness, or epigastric discomfort may predominate rather than classic chest pain.
Do not stop antiplatelet medicines without cardiologist advice; adhere to rehab & medications.